Increased levels of UCHL1 are a compensatory response to disrupted ubiquitin homeostasis in spinal muscular atrophy and do not represent a viable therapeutic target

Rachael Powis, Chantal Mutsaers, Thomas Wishart, Gillian Hunter, Brunhilde Wirth, Thomas Gillingwater

Research output: Contribution to journalArticlepeer-review

23 Citations (Scopus)

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Medicine & Life Sciences